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Pathogenicity of Pekin duck- and goose-origin parvoviruses in Pekin ducklings.
TLDR
The findings suggest that both variant and classical GPVs are pathogenic for Pekin ducklings, while the former is more virulent than the latter. Expand
Pathogenicity of a variant goose parvovirus, from short beak and dwarfism syndrome of Pekin ducks, in goose embryos and goslings
TLDR
The findings are likely to reinforce the importance of surveillance for parvoviruses in different waterfowl species and stimulate further study to elucidate the impact of mutations in the GPV genome on its pathogenicity to goslings and ducks. Expand
A duck reovirus variant with a unique deletion in the sigma C gene exhibiting high pathogenicity in Pekin ducklings.
TLDR
Findings suggest that HN5d is a novel duck reovirus isolate with severer pathogenicity in Pekin ducklings. Expand
Genetic detection and characterization of goose parvovirus: Implications for epidemiology and pathogenicity in Cherry Valley Pekin ducks.
TLDR
To understand the prevalence of GPV infection in Cherry Valley Pekin ducks, the major duck species rearing in China, tissue samples were collected between March 2015 and June 2016 and fell into the West-European linage of the GPV-related group. Expand
Isolation and detection of duck astrovirus CPH: implications for epidemiology and pathogenicity
TLDR
The detection of DAstV/CPH in 36/130 dead-in-shell duck embryo samples collected from different hatcheries located in six provinces suggests that the virus may be horizontally and vertically transmitted and associated with hatchability problems. Expand
Genetic characterization of a novel calicivirus from a goose
TLDR
Calicivirus H146 was identified as a founding member of a novel genus for which the name “Sanovirus” is proposed, similar to what has been found in the bat sapovirus TLC58. Expand
Effect of fetal calf serum on propagation of duck hepatitis A virus genotype 3 in duck embryo fibroblast cells
TLDR
Both virulent and attenuated DAHV-3 strains can establish noncytocidal, productive infections in DEF cells and FCS has an inhibitory effect on DHAv-3 replication, which may be attributed to action of a non-specific inhibitory factor present in FCS directly on the virus. Expand
Proteomics reveals the effect of type I interferon on the pathogenicity of duck hepatitis A virus genotype 3 in Pekin ducks.
TLDR
Findings support the view that the type I interferon may play an extremely important role in the pathogenic mechanism of DHAV-3, which has become the most prevalent pathogen of duck viral hepatitis in Asian duck industry in recent years. Expand
Mechanisms of Hepatocellular Injury in Hepatitis A
TLDR
This review summarizes the current knowledge of the mechanisms of hepatocellular injury in hepatitis A and suggests different mechanisms may be involved under different conditions and they are not necessarily mutually exclusive. Expand
Elevated NTCP expression by an iPSC-derived human hepatocyte maintenance medium enhances HBV infection in NTCP-reconstituted HepG2 cells
TLDR
The data suggest that this particular medium could be used to enhance HBV infection in NTCP-reconstituted hepatocytes in vitro. Expand
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