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Preparation of ZrB2-MoSi2 high oxygen resistant coating using nonequilibrium state powders by self-propagating high-temperature synthesis
To achieve high oxygen blocking structure of the ZrB2-MoSi2 coating applied on carbon structural material, ZrB2-MoSi2 coating was prepared by spark plasma sintering (SPS) method utilizing ZrB2-MoSi2…
Oxidation protection of B4C modified HfB2-SiC coating for C/C composites at 1073–1473 K
Oxidation inhibition behaviors of the HfB2-SiC-TaSi2 coating for carbon structural materials at 1700 °C
In vitro anti-inflammatory effect of picrasmalignan A by the inhibition of iNOS and COX‑2 expression in LPS‑activated macrophage RAW 264.7 cells.
- F. Zhao, L. Chen, Chenchen Bi, Menglin Zhang, Wei-Hua Jiao, X. Yao
- Biology, MedicineMolecular medicine reports
- 1 November 2013
Results showed that picrasmalignan A suppressed lipopolysaccharide-stimulated NO production and pro-inflammatory cytokine secretion, including TNF-α and IL-6, in a dose-dependent manner, and significantly inhibited the expression and enzymatic activity of iNOS and COX-2.
Combined treatment with low dose prednisone and escin improves the anti-arthritic effect in experimental arthritis.
Dexamethasone-loaded hollow hydroxyapatite microsphere promotes odontogenic differentiation of human dental pulp cells in vitro
The data suggest that sustained release of DEX from DHHAM could efficiently enhance odontogenic differentiation of hDPCs.
Inhibition of lipopolysaccharide-induced iNOS and COX-2 expression by indole alkaloid, 3-(hydroxymethyl)-6,7-dihydroindolo[2,3-a]quinolizin-(12H)-one, via NF-κB inactivation in RAW 264.7 macrophages.
It is suggested that the effect of 3-(hydroxymethyl)-6,7-dihydroindolo[2,3-a]quinolizin-(12H)-one-mediated inhibition of the expression of LPS-induced iNOS and COX-2 genes is due to the suppression of NF-κB activation in the transcriptional level.
Preparation of MoSi2-SiB6 oxidation inhibition coating on graphite by spark plasma sintering method
Sleep Quality Improvement Enhances Neuropsychological Recovery and Reduces Blood Aβ42/40 Ratio in Patients with Mild–Moderate Cognitive Impairment
This is the first clinical study on sleep quality improvement in Alzheimer’s disease patients and found that the blood Aβ42/40 ratio held the highest significance in predicting sleep disorder occurrence.
Sleep Treatment Improves Neuropsychological Symptoms and Reduces Blood Aβ42/pTau Protein in Alzheimer's Disease Patients: a Longitudinal Study
A complete recovery is critical for a full improvement of all behavioral and neuropsychological assessments, which is also associated with a deep reduction of Aβ42 and Tau-pT181 levels, which are the core biomarkers in Alzheimer’s disease.