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The authors would like to acknowledge the support received from INSEAD's R&D department and the Reginald H. Jones Center of the Wharton School. We are also grateful for the many suggestions, comments and critiques generously offered and three anonymous reviewers Of course, we remain responsible for any remaining error and omissions. In its working paper(More)
The management of technology acquisitions-acquisitions of small technology based firms by large established firms-poses an organizational paradox. Acquirers must integrate acquired firms in order to exploit their technologies in a coordinated manner; at the same time, they must preserve organizational autonomy for acquired firms in order to avoid disrupting(More)
Using a novel and comprehensive database on the performance of US and EU private equity (PE) funds and their underlying investments, we find that the performance of PE funds is comparable to public market performance. We show how sensitive this result is to various assumptions and thereby reconcile existing divergent estimates. We find evidence consistent(More)
A novel human gene, encoding a 188 amino acid polypeptide that contains a region similar to that of the epidermal growth factor, has been isolated. The gene, expressed in undifferentiated human and mouse teratocarcinoma cells, is shut off after inducing the cells to differentiate by treatment with retinoic acid. Introduction of the cDNA under the control of(More)
Using a novel and comprehensive database on both US and EU private equity funds and their underlying investments, we study the drivers of private equity fund performance. First, we study whether their hedging properties are attractive enough to justify their low performance. We document that fund performance co-varies positively with both business cycles(More)
X-linked lymphoproliferative syndrome (XLP or Duncan disease) is characterized by extreme sensitivity to Epstein-Barr virus (EBV), resulting in a complex phenotype manifested by severe or fatal infectious mononucleosis, acquired hypogammaglobulinemia and malignant lymphoma. We have identified a gene, SH2D1A, that is mutated in XLP patients and encodes a(More)