Marie Hutchinson

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BACKGROUND Although there is a sizeable body of evidence regarding the nature of hostile behaviours among clinicians in the nursing workplace, what is less clear is the nature of the relationship between these behaviours and patient care. To inform the development of appropriate intervention strategies we examine the level of evidence detailing the(More)
Mutations in the skeletal muscle channel (SCN4A), encoding the Nav1.4 voltage-gated sodium channel, are causative of a variety of muscle channelopathies, including non-dystrophic myotonias and periodic paralysis. The effects of many of these mutations on channel function have been characterized both in vitro and in vivo. However, little is known about the(More)
  • Citation Corrochano, R Männikkö, P I Joyce, P Mcgoldrick, J Wettstein, G Lassi +19 others
  • 2014
The Harvard community has made this article openly available. Please share how this access benefits you. Your story matters. et al. 2014. " Novel mutations in human and mouse SCN4A implicate AMPK in myotonia and periodic paralysis. Mutations in the skeletal muscle channel (SCN4A), encoding the Na v 1.4 voltage-gated sodium channel, are causative of a(More)
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