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Trypanosoma cruzi: parasite shed vesicles increase heart parasitism and generate an intense inflammatory response.

• Ana Claudia Trocoli Torrecilhas, Renata Rosito Tonelli, +7 authors Maria Júlia Manso Alves
• Microbes and infection
• 2009

Trypanosoma cruzi trypomastigotes continuously shed into the medium plasma membrane fragments sealed as vesicles enriched in glycoproteins of the gp85 and trans-sialidase (TS) superfamily and… (More)

Identification of Differentially Expressed Proteins from Leishmania amazonensis Associated with the Loss of Virulence of the Parasites

• Rubens Daniel Miserani Magalhães, Mariana Costa Duarte, +11 authors Eduardo Antonio Ferraz Coelho
• PLoS neglected tropical diseases
• 2014

BACKGROUND The present study analyzed whether or not the in vitro cultivation for long periods of time of pre-isolated Leishmania amazonensis from lesions of chronically infected BALB/c mice was able… (More)

1-Thio-beta-D-galactofuranosides: synthesis and evaluation as beta-D-galactofuranosidase inhibitors.

• C. Marino, Kan Marino, Luiz Claudio Miletti, Maria Júlia Manso Alves, Walter Colli, Rosa M. de Lederkremer
• Glycobiology
• 1998

Beta-D-galactofuranosidase is a good chemotherapeutic target for the design of inhibitors, since beta-D-galactofuranose is a constituent of important parasite glycoconjugates but is not present in… (More)

Scaffold proteins LACK and TRACK as potential drug targets in kinetoplastid parasites: Development of inhibitors.

• Nir Qvit, Deborah Schechtman, +19 authors Daria Mochly-Rosen
• International journal for parasitology. Drugs and…
• 2016

Parasitic diseases cause ∼ 500,000 deaths annually and remain a major challenge for therapeutic development. Using a rational design based approach, we developed peptide inhibitors with… (More)

Nitric oxide affects trypomastigote to amastigote differentiation in Trypanosoma cruzi.

• Chrislaine Oliveira Soares, Caroline Pietá Dias, Walter Colli, Maria Júlia Manso Alves
• Free radical biology & medicine
• 2014

Trypanosoma cruzi is the etiologic agent of Chagas disease. Two main distinct forms are present in the mammalian host: trypomastigote, an infective form, and the amastigote, a typical replicative… (More)

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