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Association of the human minK gene 38G allele with atrial fibrillation: evidence of possible genetic control on the pathogenesis of atrial fibrillation.
TLDR
The association between the minK 38G allele and clinical atrial fibrillation is reported and suggest possible genetic control on the pathogenesis of atrialfibrillation.
Changes in the mRNA Levels of Delayed Rectifier Potassium Channels in Human Atrial Fibrillation
TLDR
Because the decrease in potassium currents results in a prolonged action potential, the shortening of the atrial effective refractory period in atrial fibrillation should be attributed to other factors.
Effect of hypercholesterolemia on myocardial function in New Zealand white rabbits.
TLDR
It is concluded that hypercholesterolemia results in suppression of the maximal contractile function and in a longer systolic contractile time course.
Activation of the transient receptor potential M2 channel and poly(ADP-ribose) polymerase is involved in oxidative stress-induced cardiomyocyte death
TLDR
It is shown that H2O2 activates both apoptotic and necrotic machineries in the same myocyte and that the ultrastructure seen using EM is very similar to that in I/R animal studies.
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