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Association of the human minK gene 38G allele with atrial fibrillation: evidence of possible genetic control on the pathogenesis of atrial fibrillation.
The association between the minK 38G allele and clinical atrial fibrillation is reported and suggest possible genetic control on the pathogenesis of atrialfibrillation.
Cardioprotective effect of resveratrol, a natural antioxidant derived from grapes.
Down-regulation of L-type calcium channel and sarcoplasmic reticular Ca(2+)-ATPase mRNA in human atrial fibrillation without significant change in the mRNA of ryanodine receptor, calsequestrin and…
Resveratrol protects myocardial ischemia-reperfusion injury through both NO-dependent and NO-independent mechanisms.
Phosphatidylinositol-3-kinase is involved in the antihyperglycemic effect induced by resveratrol in streptozotocin-induced diabetic rats.
Changes in the mRNA Levels of Delayed Rectifier Potassium Channels in Human Atrial Fibrillation
Because the decrease in potassium currents results in a prolonged action potential, the shortening of the atrial effective refractory period in atrial fibrillation should be attributed to other factors.
Resveratrol enhances insulin secretion by blocking K(ATP) and K(V) channels of beta cells.
Effect of hypercholesterolemia on myocardial function in New Zealand white rabbits.
It is concluded that hypercholesterolemia results in suppression of the maximal contractile function and in a longer systolic contractile time course.
Activation of the transient receptor potential M2 channel and poly(ADP-ribose) polymerase is involved in oxidative stress-induced cardiomyocyte death
It is shown that H2O2 activates both apoptotic and necrotic machineries in the same myocyte and that the ultrastructure seen using EM is very similar to that in I/R animal studies.