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Locations of adenovirus genes required for the replication of adenovirus-associated virus.
It is demonstrated that three restriction endonuclease fragments of adenovirus DNA are necessary for production of infectious AAV in 293-31 cells (an adanovirus type 5-transformed human embryonic kidney cell line) and additional experiments indicate, however, that early region 1A provides an essential function(s) for AAV replication, whereasEarly region 1B probably does not.
Efficient synthesis of adeno-associated virus structural proteins requires both adenovirus DNA binding protein and VA I RNA.
Human B cells express IL-5 receptor messenger ribonucleic acid and respond to IL-5 with enhanced IgM production after mitogenic stimulation with Moraxella catarrhalis.
- M. M. Huston, J. Moore, H. J. Mettes, G. Tavana, D. Huston
- Biology, MedicineJournal of immunology
- 15 February 1996
It is demonstrated that, as with murine B cells, human B cells express message for IL-5R but can respond toIL-5 only if appropriately stimulated to undergo terminal differentiation.
Production of IL-5 and granulocyte-macrophage colony-stimulating factor by naive human mast cells activated by high-affinity IgE receptor ligation.
Mitochondria control expression of a murine cell surface antigen
It is reported here that Mta expression in somatic cell hybrids requires functional mitochondria from the Mta+ parent cell line, the first evidence for mitochondrial control of the expression of a cell membrane molecule in eukaryotes.
Adeno-associated virus proteins: origin of the capsid components
It is demonstrated definitively that these proteins are encoded in the right half of the adeno-associated virus 2 genome, and one or both of the smallestAdeno- associated RNA species (2.3- or 2.6-kilobase RNA) account for their synthesis.
Mitochondrial modulation of maternally transmitted antigen: analysis of cell hybrids.
- M. M. Huston, R. Smith, R. Hull, D. Huston, R. Rich
- BiologyProceedings of the National Academy of Sciences…
- 1 May 1985
The data support the remarkable conclusion that mitochondria modulate the phenotypic expression of a cell surface molecule.
Enhanced detection of human IL-5 in biological fluids utilizing murine monoclonal antibodies which delineate distinct neutralizing epitopes.
Immunoglobulin deficiency syndromes and therapy.