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Clinical Toxicology of Anticholinesterase Agents in Humans
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The pathogenesis of organophosphate polyneuropathy.
  • M. Lotti
  • Biology
    Critical reviews in toxicology
  • 1991
This review discusses the facts regarding organophosphate-induced delayed polyneuropathy (OPIDP) as they are related to its pathogenesis rather than being a comprehensive review of all available…
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Organophosphate-Induced Delayed Polyneuropathy
TLDR
This article mainly discusses OP pesticide poisoning, particularly when caused by chlorpyrifos, dichlorvos, isofenphos, methamidophos, mipafox, trichlorfon,trichlornat, phosphamidon/mevinphos and by certain carbamates.
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Cholinesterase inhibition: complexities in interpretation.
  • M. Lotti
  • Biology, Medicine
    Clinical chemistry
  • 1 December 1995
TLDR
Assessment of the relation between inhibition of erythrocytes and nervous tissue AChE depends on the pharmacokinetics of inhibitors, and pharmacodynamic factors such as spontaneous reactivation and aging of inhibited enzyme should also be considered.
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Poisoning by organophosphorus insecticides and sensory neuropathy
  • A. Moretto, M. Lotti
  • Medicine, Biology
    Journal of neurology, neurosurgery, and…
  • 1 April 1998
TLDR
A sensory-motor polyneuropathy caused by organophosphate insecticides might occur after a severe poisoning and the sensory component, if present, is milder than the motor one.
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Organophosphorus and carbamate insecticide poisoning.
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Promotion of organophosphate-induced delayed polyneuropathy by phenylmethanesulfonyl fluoride.
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Neurotoxicity of organophosphorus pesticides: predictions can be based on in vitro studies with hen and human enzymes
TLDR
Comparisons of the in vitro effects seen with hen and human enzymes facilitates extrapolations to the human in vivo situation.
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Inhibition of lymphocytic neuropathy target esterase predicts the development of organophosphate-induced delayed polyneuropathy
TLDR
Measurement of lymphocytic NTE might be used as a clinical test to predict the development of OP-induced delayed polyneuropathy.
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Individual exposure to particulate matter and the short-term arrhythmic and autonomic profiles in patients with myocardial infarction.
TLDR
Exposure to ultrafine particles is associated with autonomic dysregulation in selected patients with myocardial infarction and the underlying mechanisms remain hypothetical because inflammation may be evoked by PM or be related to the disease itself.
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