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Chronic rejection.
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Brain death and its influence on donor organ quality and outcome after transplantation.
TLDR
In virtually all experimental studies of organ transplantation, young, healthy living animals are used as donors; in clinical practice, in contrast, a relatively low percentage of organs comes from living donors, as cadavers remain the primary source of supply.
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Influence of donor brain death on chronic rejection of renal transplants in rats.
TLDR
Donor BD accelerates the progression of long-term changes associated with kidney Tx and is an important risk factor for chronic rejection, which explains in part the clinically noted difference in long- term function between organs from cadaver and living sources.
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Increased urinary neutrophil gelatinase associated lipocalin levels in a rat model of upper urinary tract infection.
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Genomewide Expression Profiles of Rat Model Renal Isografts From Brain Dead Donors
Background. It has been well documented that two factors, brain death (BD) and ischemia/reperfusion (I/R) injury, have distinct but overlapping adverse influences on the clinical outcome of renal…
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Activation of the Heart by Donor Brain Death Accelerates Acute Rejection After Transplantation
TLDR
Donor brain death is deleterious to transplanted hearts because the resultant upregulation of inflammatory factors provokes host immune mechanisms and accelerates the acute rejection process in unmodified hosts.
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Accelerated Rejection of Renal Allografts From Brain-Dead Donors
TLDR
Donor brain death is a significant risk factor for peripheral organs used for transplantation because the activated state of such organs appears to trigger host immune mechanisms that accelerate the process of acute rejection.
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Activation of inflammatory mediators in rat renal isografts by donor brain death.
TLDR
The intense nonimmune inflammation produced in isografts after donor BD may represent the initial stages of a continuum between an initial nonspecific and later immune reactivity, when placed in the context of allotransplantation.
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A model of gradual onset brain death for transplant-associated studies in rats.
TLDR
A controlled model of gradual onset brain death in the rat in which normotension can be sustained for several hours before the kidneys are removed for transplantation, suitable for transplant-related studies involving organs from donors with irreversible central injury.
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Serum Neutrophil Gelatinase-Associated Lipocalin as a Predictor of Organ Recovery from Delayed Graft Function after Kidney Transplantation from Donors after Cardiac Death
TLDR
Monitoring of serum NGAL levels may allow us to predict graft recovery and the need for HD after a KTx from a DCD, and to predict early functional recovery of transplanted DCD kidneys.
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