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Silent period evoked by transcranial stimulation of the human cortex and cervicomedullary junction.
The findings indicate that the first 50 ms of the silent period after TCS are produced mainly by spinal mechanisms such as after‐hyperpolarization and recurrent inhibition of the spinal motoneurones and if descending inhibitory fibres contribute, their contribution is small.
Brain–computer interface boosts motor imagery practice during stroke recovery
Brain–computer interfaces (BCIs) can provide instantaneous and quantitative measure of cerebral functions modulated by MI, and the efficacy of BCI‐monitored MI practice as add‐on intervention to usual rehabilitation care was evaluated in subacute stroke patients.
Facilitation of muscle evoked responses after repetitive cortical stimulation in man
RTMS of these parameters increases the excitability of the motor cortex and that this effect outlasts the train for almost 1 s, and at the spinal level, rTMS may increase presynaptic inhibition of Ia afferent fibers responsible for the H-reflex.
Effects of diazepam, baclofen and thiopental on the silent period evoked by transcranial magnetic stimulation in humans
- M. Inghilleri, A. Berardelli, P. Marchetti, M. Manfredi
- Biology, PsychologyExperimental Brain Research
- 1 June 1996
Findings show that the cortical silent period can be modified pharmacologically, and Diazepam possibly shortens the silent period by modulating GABA A receptors at a subcortical site.
Genome-wide Analyses Identify KIF5A as a Novel ALS Gene
Safety and efficacy of eculizumab in anti-acetylcholine receptor antibody-positive refractory generalised myasthenia gravis (REGAIN): a phase 3, randomised, double-blind, placebo-controlled,…
Silent period in upper limb muscles after noxious cutaneous stimulation in man.
Functional organization of the trigeminal motor system in man. A neurophysiological study.
- G. Cruccu, A. Berardelli, M. Inghilleri, M. Manfredi
- BiologyBrain : a journal of neurology
- 1 October 1989
TCS in intact human subjects was used to investigate the corticobulbar projections and the functional organization of the trigeminal motor system, finding that the masseter inhibitory period after magnetic TCS may be due to excitation of corticofugal inhibitory fibres and to mechanical activation of Golgi tendon organs.
Ovarian hormones and cortical excitability. An rTMS study in humans