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Nutritional stimulation of cholecystokinin receptors inhibits inflammation via the vagus nerve
The immune system in vertebrates senses exogenous and endogenous danger signals by way of complex cellular and humoral processes, and responds with an inflammatory reaction to combat putativeExpand
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Macrophage inhibitory factor, plasminogen activator inhibitor-1, other acute phase proteins, and inflammatory mediators normalize as a result of weight loss in morbidly obese subjects treated with
Obesity is demonstrated to be associated with an enhanced inflammatory state, which is suggested to be a cause for the development of obesity-related morbidity. It was hypothesized that a decrease inExpand
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N‐acetyl resonances in in vivo and in vitro NMR spectroscopy of cystic ovarian tumors
An unassigned and prominent resonance in the region from δ 2.0–2.1 ppm has frequently been found in the in vivo MR spectra of cancer patients. We demonstrated the presence of this resonance with inExpand
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Lipid-Rich Enteral Nutrition Reduces Postoperative Ileus in Rats via Activation of Cholecystokinin-Receptors
Objective:This study investigates the effect of lipid-rich nutrition on the local inflammatory response and gastrointestinal hypomotility in a rat model of postoperative ileus.Expand
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Strain-Specific Effects of Probiotics on Gut Barrier Integrity following Hemorrhagic Shock
ABSTRACT Probiotic therapy modulates the composition of the intestinal flora and inhibits the inflammatory response. These properties may be of benefit in the preservation of gut barrier integrityExpand
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Cholecystokinin/Cholecystokinin-1 Receptor-Mediated Peripheral Activation of the Afferent Vagus by Enteral Nutrients Attenuates Inflammation in Rats
Objective:The current study investigates activation of the nutritional anti-inflammatory pathway by lipid-rich nutrition. Background:Enteral nutrition activates humoral and neural pathways toExpand
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Starvation compromises Paneth cells.
Lack of enteral feeding, with or without parenteral nutritional support, is associated with increased intestinal permeability and translocation of bacteria. Such translocation is thought to beExpand
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Pretreatment With High-Fat Enteral Nutrition Reduces Endotoxin and Tumor Necrosis Factor-&agr; and Preserves Gut Barrier Function Early After Hemorrhagic Shock
Gram-negative sepsis is a potentially fatal clinical syndrome characterized by a proinflammatory response (tumor necrosis factor-&agr;) to bacterial (endo)toxins and gut barrier function loss.Expand
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INTESTINAL CYTOSKELETON DEGRADATION PRECEDES TIGHT JUNCTION LOSS FOLLOWING HEMORRHAGIC SHOCK
Hemorrhagic shock (HS) leads to intestinal barrier loss, causing systemic inflammation, which in turn can ultimately lead to multiorgan dysfunction syndrome. Barrier function is based on tightExpand
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Postshock Intervention With High-Lipid Enteral Nutrition Reduces Inflammation and Tissue Damage
Objective:To investigate the effects of high-lipid enteral nutrition in a setting of developing inflammation and tissue damage. Background:An excessive inflammatory response following severe traumaExpand
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