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Salsolinol is a Putative Endogenous Neuro‐intermediate Lobe Prolactin‐Releasing Factor
The data clearly suggest that SAL is synthesized in situ and this compound can play a role in the regulation of pituitary prolactin secretion.
Binding site of salsolinol: its properties in different regions of the brain and the pituitary gland of the rat
Distribution of norepinephrine and dopamine in cerebral cortical areas of the rat
Anxiolytic 2,3-benzodiazepines, their specific binding to the basal ganglia
Dopamine transporters participate in the physiological regulation of prolactin.
Evidence is provided that DATs play a physiological role in the regulation of DA release from and TH expression in NEDA neurons and consequently PRL secretion and PRL gene expression and further support the previous observation that theregulation ofPRL secretion involves all three populations of NEDa neurons.
Effects of reserpine and antidepressants on dopamine and DOPAC (3,4-dihydroxyphenylacetic acid) concentrations in the striatum, olfactory tubercle and median eminence of rats.
Noradrenergic innervation of the rat hypothalamus: Experimental biochemical and electron microscopic studies
Stress‐ as Well as Suckling‐Induced Prolactin Release is Blocked by a Structural Analogue of the Putative Hypophysiotrophic Prolactin‐Releasing Factor, Salsolinol
1MeDIQ profoundly inhibited suckling‐, immobilization‐, as well as formalin‐stress induced prolactin release without any influence on corticosterone secretion, suggesting that salsolinol can play a pivotal role in the regulation of prolactIn release induced by either physiological (suckling) or environmental (stress) stimuli.
A novel specific binding site for homophthalazines in the rat brain.