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Insulin secretory defects in polycystic ovary syndrome. Relationship to insulin sensitivity and family history of non-insulin-dependent diabetes mellitus.
The increased prevalence of non-insulin-dependent diabetes mellitus (NIDDM) among women with polycystic ovary syndrome (PCOS) has been ascribed to the insulin resistance characteristic of PCOS. ThisExpand
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Demonstration of rapid light-induced advances and delays of the human circadian clock using hormonal phase markers.
To determine the magnitude and direction of phase shifts of human circadian rhythms occurring within 1 day after a single exposure to bright light, plasma thyrotropin, melatonin, and cortisol levelsExpand
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Insulin secretory abnormalities in subjects with hyperglycemia due to glucokinase mutations.
Pancreatic beta-cell function was studied in six subjects with mutations in the enzyme glucokinase (GCK) who were found to have elevated fasting and postprandial glucose levels in comparison to sixExpand
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Treatment with the oral antidiabetic agent troglitazone improves beta cell responses to glucose in subjects with impaired glucose tolerance.
Impaired glucose tolerance (IGT) is associated with defects in both insulin secretion and action and carries a high risk for conversion to non-insulin-dependent diabetes mellitus (NIDDM).Expand
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Nocturnal exercise phase delays circadian rhythms of melatonin and thyrotropin and thyrotropin secretion in normal men
To determine whether a single episode of physical activity is capable of inducing rapid phase shifts in human circadian rhythms, 17 subjects were studied two times under constant routine conditions,Expand
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Nocturnal exercise phase delays circadian rhythms of melatonin and thyrotropin secretion in normal men.
To determine whether a single episode of physical activity is capable of inducing rapid phase shifts in human circadian rhythms, 17 subjects were studied two times under constant routine conditions,Expand
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Insulin secretion and clearance during low-dose graded glucose infusion.
The present study was undertaken in normal volunteers to define the alterations in beta-cell responsiveness to glucose associated with different physiological states, including fasting and refeeding,Expand
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Relationships between sleep quality and glucose regulation in normal humans.
To define the effects of sleep on glucose regulation, we analyzed plasma glucose levels, insulin secretion rates (ISR), and plasma growth hormone and cortisol levels in normal subjects receiving aExpand
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GLP-1-induced alterations in the glucose-stimulated insulin secretory dose-response curve.
The present study was undertaken to establish in normal volunteers the alterations in beta-cell responsiveness to glucose associated with a constant infusion of glucagon-like peptide-1 (GLP-1) or aExpand
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