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Prevalence of endometriosis in adolescent girls with chronic pelvic pain not responding to conventional therapy.
- M. Laufer, L. Goitein, M. Bush, D. Cramer, S. Emans
- Journal of pediatric and adolescent gynecology
- 1 November 1997
Adolescents with chronic pelvic pain not responding to medical therapy have a high rate of endometriosis and should be referred to a gynecologist who is experienced with the subtle laparoscopic findings of atypical endometRIosis to diagnose the etiology of the pelvic pain and initiate appropriate therapy. Expand
Azithromycin and Erythromycin in the Treatment of Cervical Chlamydial Infection During Pregnancy
Objective To compare azithromycin and erythromycin in regard to side effects, intolerance, and cure rate in a pregnant population with chlamydial cervicitis. Methods Thirty women were randomized to… Expand
Pregnancy outcomes from more than 1,800 in vitro fertilization cycles with the use of 24-chromosome single-nucleotide polymorphism-based preimplantation genetic testing for aneuploidy.
The findings that SNP- based PGT-A can mitigate the negative effects of maternal age on IVF outcomes in cycles with transfer, and that pregnancy outcomes from SET cycles are not significantly different from those of double-embryo transfer cycles, support the use of SET when transfers are combined with SNP-based P GT-A. Expand
Hematosalpinx with pelvic pain after endometrial ablation confirms the postablation-tubal sterilization syndrome.
A 39-year-old woman with previous tubal ligation underwent laparoscopic-assisted vaginal hysterectomy for debilitating pelvic pain 1 year after endometrial ablation, and symptoms and findings confirm the postablation-tubal sterilization syndrome. Expand
Patients’ administration preferences: progesterone vaginal insert (Endometrin®) compared to intramuscular progesterone for Luteal phase support
- A. Beltsos, Mark D. Sanchez, K. Doody, M. Bush, A. Domar, M. Collins
- Reproductive Health
- 11 November 2014
It is suggested that PVI provides an easy-to-use and convenient method for providing the necessary luteal phase support for IVF cycles without the pain and inconvenience of daily IM PIO. Expand
The role of fallopian tube anastomosis in training fellows: a survey of current reproductive endocrinology fellows and practitioners.
- A. Armstrong, A. Neithardt, R. Alvero, F. Sharara, M. Bush, J. Segars
- Fertility and sterility
- 1 August 2004
This survey of fellows and reproductive endocrinologists in practice suggests that decreasing numbers of tubal reanastomosis procedures are being performed. This change has occurred both in fellow… Expand
Clinical comparison of ovarian stimulation and luteal support agents in patients undergoing GnRH antagonist IVF cycles.
- Charles E. Miller, E. Zbella, B. Webster, K. Doody, M. Bush, M. Collins
- The Journal of reproductive medicine
- 1 March 2013
In this study HP-hMG and rhFSH were equally effective for ovarian stimulation during GnRH antagonist IVF cycles and both PVI and PIO are viable options for luteal support. Expand
Treatment with gonadotropin-releasing hormone (GnRH) antagonists in women suppressed with GnRH agonist may avoid cycle cancellation in patients at risk for ovarian hyperstimulation syndrome.
Forty-seven patients at high risk for ovarian hyperstimulation syndrome because of markedly elevated serum E2 levels on either long-luteal or microdose flare leuprolide acetate regimens were treated… Expand
E2-induced degradation of uterine insulin receptor substrate-2: requirement for an IGF-I-stimulated, proteasome-dependent pathway.
- R. Richards, D. M. Klotz, M. Bush, D. Walmer, R. Diaugustine
- Biology, Medicine
- 1 September 2001
The data suggest that the E2-induced decrease in uterine insulin receptors substrate-2 requires IGF-I signaling, is not dependent solely on insulin receptor substrate-1 and PI3K, and is blocked by progesterone as well as by pharmacological inhibition of proteasomal protease activity. Expand
Evidence of Juxtacrine Signaling for Transforming Growth Factor α inHuman Endometrium
Data are consistent with a juxtacrine mode of signaling for TGFalpha between endometrial cells, and between the luminal surface epithelium and preimplantation embryos, since the EGFR is present in the endometrium and on the surface of embryos. Expand