Lisa H. Kriener

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signaling − 2 channel activity and lung fluid balance in vivo via O Nadph oxidase regulates alveolar epithelial sodium You might find this additional info useful... sodium channels to promote alveolar fluid clearance in vivo-Adrenergic agonists differentially regulate highly selective and nonselective epithelial β including high resolution figures, can be(More)
β-Adrenergic receptors (β-AR) increase epithelial sodium channel (ENaC) activity to promote lung fluid clearance. However, the effect of selective β-AR agonist on highly selective cation (HSC) channels or nonselective cation (NSC) channels in alveolar type 1 (T1) and type 2 (T2) cells is unknown. We hypothesized that stimulation with β(1)-AR agonist(More)
Episodic ataxia type 2 (EA2) is an autosomal dominant disorder associated with attacks of ataxia that are typically precipitated by stress, ethanol, caffeine or exercise. EA2 is caused by loss-of-function mutations in the CACNA1A gene, which encodes the α1A subunit of the CaV2.1 voltage-gated Ca(2+) channel. To better understand the pathomechanisms of this(More)
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