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OBJECTIVES To investigate if the reduced P50, N100 and P200 auditory evoked potential (EP) components and gating deficits seen in schizophrenia can be explained in terms of response incompleteness. METHODS Twenty-five healthy and schizophrenia participants were studied using pairs of 1000Hz tones (S1 and S2, 0.5s apart) separated by 8.0s. A(More)
In contrast to sensory gating, the brain's ability to re-respond to relevant stimuli and the potential differences between healthy and schizophrenic participants have not been studied in great detail. Here, we explore what auditory paradigms are useful to measure this re-responding ability. Evoked potentials (EPs) were obtained from the Cz channel using 3(More)
An unsupervised correlation-based clustering method was developed to assess the trial-to-trial variability of auditory evoked potentials (AEPs). The method first decomposes single trials into three frequency bands, each containing activity primarily associated with one of the three major AEP components, i.e., P50, N100 and P200. Next, single-trial evoked(More)
The mid-latency auditory evoked response (MLAER) consists of the P50, N100 and P200 components. P50 is widely used to examine sensory gating. There is growing evidence that phase-reorganization of the oscillatory components comprising EEG activity, and especially in the 4-8 Hz (theta) band, is responsible for MLAER generation. Consequently, all or part of(More)
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