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Analysis of ISG expression in chronic hepatitis C identifies viperin as a potential antiviral effector
Interferon (IFN) α inhibits hepatitis C virus (HCV) replication both clinically and in vitro; however, the complete spectrum of interferon‐stimulated genes (ISGs) expressed in the HCV‐infected liverExpand
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Long-term persistence of Coxiella burnetii after acute primary Q fever.
BACKGROUND Long-term persistence of C. burnetii in infected animals was established in the 1950s and 60s, but the implications for human Q fever are not fully explored. AIM To compare theExpand
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Alcohol metabolism increases the replication of hepatitis C virus and attenuates the antiviral action of interferon.
The interactions between hepatitis C virus (HCV) and alcohol metabolism are not well understood. To determine the effect that alcohol metabolism has on HCV replication and the antiviral action ofExpand
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L-Proline induces differentiation of ES cells: a novel role for an amino acid in the regulation of pluripotent cells in culture.
The development of cell therapeutics from embryonic stem (ES) cells will require technologies that direct cell differentiation to specific somatic cell lineages in response to defined factors. TheExpand
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Expression of the CXCR3 ligand I‐TAC by hepatocytes in chronic hepatitis C and its correlation with hepatic inflammation
The factors that regulate lymphocyte traffic in chronic hepatitis C (CHC) are not completely defined. Interferon (IFN)‐inducible T cell α chemoattractant (I‐TAC) is a relatively new member of theExpand
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Q fever: still a mysterious disease.
Sir, In the concluding paragraph of his editorial,1 Dr Raoult makes a statement about the dangers of false‐positive PCR assays arising from DNA contamination during the detection of CoxiellaExpand
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Q Fever Research Group (QRG), Adelaide: Activities‐Exit Summary 1980–2004
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