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Phylogenetic Relationships and Coaggregation Ability of Freshwater Biofilm Bacteria
- A. Rickard, S. Leach, L. Hall, Clive M. Buswell, N. High, P. Handley
- Biology, Medicine
- Applied and Environmental Microbiology
- 1 July 2002
ABSTRACT Nineteen numerically dominant heterotrophic bacteria from a freshwater biofilm were identified by 16S ribosomal DNA gene sequencing, and their coaggregation partnerships were determined.… Expand
Fatty acids stimulate cholecystokinin secretion via an acyl chain length‐specific, Ca2+‐dependent mechanism in the enteroendocrine cell line STC‐1
- J. McLaughlin, R. Lomax, L. Hall, G. Dockray, D. Thompson, G. Warhurst
- Biology, Medicine
- The Journal of physiology
- 1 November 1998
1 The present study has investigated whether fatty acids directly influence peptide release from enteroendocrine cells using STC‐1, a mouse intestinal endocrine tumour cell line, previously shown to… Expand
Use of coculture of colonic mucosal biopsies to investigate the release of eicosanoids by inflamed and uninflamed mucosa from patients with inflammatory bowel disease.
Eicosanoid production was measured in cultured biopsies of colonic mucosa from control patients, with the irritable bowel syndrome, and from patients with proctosigmoiditis and with colonic Crohn's… Expand
Platelet activating factor: release from colonic mucosa in patients with ulcerative colitis and its effect on colonic secretion.
Inflammatory mediators have been implicated in the pathophysiology of ulcerative colitis. They may stimulate intestinal secretion and contribute to the production of diarrhoea. Platelet activating… Expand
The influence of a parasympathetic agonist and antagonist on human intestinal transport in vivo.
Abstract We studied the influence of parasympathetic stimulation and blockade on transmucosal transport in the human jejunum and ileum in vivo. Using a triple lumen tube perfusion technique, we… Expand
Inter-relationships between inflammatory mediators released from colonic mucosa in ulcerative colitis and their effects on colonic secretion.
Metabolites of arachidonic acid have been implicated in the pathophysiology of ulcerative colitis-they can stimulate intestinal secretion, increase mucosal blood flow, and influence smooth muscle… Expand