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Two components of long-term potentiation induced by different patterns of afferent activation
TLDR
It is reported that a component of LTP not requiring the activation of NMDA receptors can be induced in area CA1, and is suppressed by an antagonist of voltage-dependent Ca2+ channels.
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Neurotrophin expression in rat hippocampal slices: A stimulus paradigm inducing LTP in CA1 evokes increases in BDNF and NT-3 mRNAs
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LTP in hippocampal area CA1 is induced by burst stimulation over a broad frequency range centered around delta.
TLDR
The data show that CA1 synapses are more broadly tuned for potentiation than previously appreciated, and N-methyl-d-aspartic acid receptors were more important for LTP induction by burst stimulation than L-type calcium channels, and NMDA receptors were even more critical for short-duration potentiation.
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Blockade of GABAA receptors facilitates induction of NMDA receptor-independent long-term potentiation.
TLDR
Increased action potential firing can at least partially account for the facilitation of NMDA receptor-independent LTP caused by blockade of GABAA receptors, consistent with prior studies demonstrating that action potentials are particularly effective stimuli for the gating of VDCCs in CA1 pyramidal cell dendrites.
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Associative learning changes intrinsic to Hermissenda type A photoreceptors.
TLDR
Type A photoreceptors also appear to be a primary site for associative information storage in Hermissenda and it is suggested that enhancement of IK-Ca by associative training may contribute to the diminished light response of Type A photoresceptors.
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Hyperpolarizing and depolarizing GABAA receptor-mediated dendritic inhibition in area CA1 of the rat hippocampus.
TLDR
The results suggest that fast field potentials are GABAA receptor-mediated population IPSPs (GABAA, fast pIPSPs) and that late field Potentials are GABAB receptor- mediated population IPS Ps (GABAB, late pIPSs).
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Role of HCO3- ions in depolarizing GABAA receptor-mediated responses in pyramidal cells of rat hippocampus.
TLDR
The sensitivity of GABAA receptor-mediated depolarizations to HCO(3-)-free medium can be explained by a decrease in presynaptic excitability and an increased resting conductance in postsynaptic neurons.
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REM sleep deprivation inhibits LTP in vivo in area CA1 of rat hippocampus
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Evidence for postsynaptic induction and expression of NMDA receptor independent LTP.
  • L. Grover
  • Biology
    Journal of neurophysiology
  • 1 March 1998
TLDR
There is a strong case for the postsynaptic induction of this form of LTP, which is found for only the AMPA-receptor-mediated EPSPs, and evidence for post Synaptic expression of NMDA receptor independent LTP.
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The mechanism of biphasic GABA responses
The neurotransmitter y-aminobutyric acid (GABA) mediates much of the inhibitory synaptic transmission in the brain. When GABA^ receptors are activated at inhibitory synapses, or when a small amount…
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