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Increases in oxidized low-density lipoprotein and other inflammatory and adhesion molecules with a concomitant decrease in high-density lipoprotein in the individuals exposed to arsenic in Bangladesh.
TLDR
All the observed associations may be the major features of arsenic exposure-related atherosclerosis leading to CVD. Expand
Interaction between chronic arsenic exposure via drinking water and plasma lactate dehydrogenase activity.
TLDR
LDH activity was found to be increased in the higher exposure groups of water and hair arsenic concentrations, and the elevated plasma LDH activity might be helpful for the early prognosis of organ or tissue damage in the individuals who were exposed to arsenic chronically in Bangladesh. Expand
Dose-response relationship between arsenic exposure and the serum enzymes for liver function tests in the individuals exposed to arsenic: a cross sectional study in Bangladesh
TLDR
Elevated serum hepatic enzyme activities in the higher exposure gradients provided new insights into arsenic- induced liver toxicity that might be helpful for the early prognosis of arsenic-induced liver diseases. Expand
Associations of total arsenic in drinking water, hair and nails with serum vascular endothelial growth factor in arsenic-endemic individuals in Bangladesh.
TLDR
Evidence is provided that arsenic exposure has a pro-angiogenic effect on humans, which may be implicated in arsenic-induced tumorigenesis and vascular diseases. Expand
Elevated levels of plasma uric acid and its relation to hypertension in arsenic-endemic human individuals in Bangladesh.
TLDR
The results suggest that arsenic exposure-related elevation of PUA levels may be implicated in arsenic-induced CVDs. Expand
Protective effects of Moringa oleifera Lam. leaves against arsenic-induced toxicity in mice.
TLDR
Food supplementation of M. oleifera leaves abrogated the arsenic-induced elevation of triglyceride, glucose, urea and the activities of alkaline phospatase, aspartate aminotransferase and alanine aminotsferase in serum. Expand
Protective effects of the dietary supplementation of turmeric (Curcuma longa L.) on sodium arsenite-induced biochemical perturbation in mice.
TLDR
Turmeric abrogated the Sa-induced elevation of serum urea, glucose, triglyceride level and alanine aminotransferase (ALT) activity except the activity of alkaline phosphatase (ALP) and prevented theSa-induced perturbation of serum butyryl cholinesterase activity (BChE). Expand
Association between arsenic exposure and plasma cholinesterase activity: a population based study in Bangladesh
TLDR
A significant inverse relationship between arsenic exposure and PChE activity in a human population in Bangladesh is found which may explain one of the biological mechanisms through which arsenic exerts its neuro-and hepatotoxicity in humans. Expand
Elevated levels of plasma Big endothelin-1 and its relation to hypertension and skin lesions in individuals exposed to arsenic.
TLDR
A novel dose-response relationship between As exposure and plasma Big ET-1 levels is demonstrated indicating the possible involvement of plasma BigET1 levels in As-induced hypertension and skin lesions. Expand
Phenylarsine oxide (PAO) induces apoptosis in HepG2 cells via ROS-mediated mitochondria and ER-stress dependent signaling pathways.
TLDR
Results showed that PAO not only had a potent inhibitory effect on the proliferation of HepG2 cells but also activated apoptosis-related proteins (e.g., caspase-3 and -9 and poly-ADP ribose polymerase) in a dose- and time-dependent manner. Expand
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