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CD152 (CTLA-4) Determines the Unequal Resistance of Th1 and Th2 Cells against Activation-induced Cell Death by a Mechanism Requiring PI3 Kinase Function
Survival of antigen-experienced T cells is essential for the generation of adaptive immune responses. Here, we show that the genetic and antibody-mediated inactivation of CD152 (cytotoxic TExpand
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Transcription factor AP-2alpha triggers apoptosis in cardiac myocytes.
Idiopathic-dilated cardiomyopathy (IDC) is a common primary myocardial disease of unknown etiology associated with apoptosis, cardiac dilatation, progressive heart failure and increased mortality. AnExpand
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Induced proximity model attracts NF-kappaB researchers.
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[Oxygen radicals as second messengers of gene expression].
Caffeine-Inhibitable Event Control but Caused by the Lack of Caspase-3 and a Ionizing Radiation Is Independent of p 53 and Cell Cycle Apoptosis Resistance of MCF-7 Breast Carcinoma Cells to
We have shown previously that ionizing radiation (IR) induces a persistent G2-M arrest but not cell death in MCF-7 breast carcinoma cells that harbor functional p53 but lack caspase-3. In the presentExpand