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Pathological and physiological identification of race C of Bipolaris maydis in China.
Purification and characterization of reversible sperm motility inhibitors from porcine seminal plasma.
Two sperm motility inhibitors (SMI1 and SMI2) were purified from porcine seminal plasma with high performance liquid chromatography (HPLC) and have similar amino acid composition, suggesting that they may be structurally related.
Use of rubella virus E1 fusion proteins for detection of rubella virus antibodies
- W. Starkey, J. Newcombe, K. Corbett, K. M. Liu, P. Sanders, J. Best
- Biology, MedicineJournal of clinical microbiology
- 1 February 1995
Two glutathione S-transferase fusion proteins containing 44 (p1503) and 75 (p1509) amino acid residues of the rubella virus E1 glycoprotein were expressed in Escherichia coli with the aim of…
Determination of in Vitro Biotoxicity in Exhaust Particulate Matter from Heavy-Duty Diesel Engine
- C. Song, R. J. Huang, Y. Wang, K. M. Liu, S. Dong, S. Dai
- Environmental Science, MedicineBulletin of environmental contamination and…
No abstract available.
Three‐dimensional analysis of vacuoles and surface invaginations of capillary endothelia in the eel rete mirabile
Three‐dimensional information from serial sections is required to understand the organization and functions of vacuole‐like structures in capillary endothelium.
Reaction of different male-sterile cytoplasm subgroups of the C group maize to the infection of Bipolaris maydis race C.
Studies on antigenic variation of types I and III poliovirus using neutralizing monoclonal antibody.
Studies on muntiacus muntjak cells infected with poliovirus.
It is shown that poliovirus could produce a strong cell genetic effect and leads to nonkilling-cell infection in Muntiacus muntjak cells.
Preliminary studies on antigenic variation of poliovirus using neutralizing monoclonal antibodies.
Cross-neutralization assays showed that strain P3/Yunnan/2/84, which was isolated from cases of poliomyelitis in a local epidemic in the Yunnan province of China in 1984, and strain P 3/Finland/23127/84 were both antigenically different from the Sabin-3 strain and the reference virulent strain.