K. Kurahashi

Publications161
h-index23
Citations1,724
Highly Influential Citations33
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Isolation and characterization of ovokinin, a bradykinin B1 agonist peptide derived from ovalbumin
Endothelium-dependent contraction induced by acetylcholine in isolated rat renal arteries.
Endothelium‐dependent contraction in intrapulmonary arteries: mediation by endothelial NK1 receptors and TXA2
TLDR
In conclusion, SP at 10−8 m induces EDC via endothelial NK1 receptors and TXA2 production, and SP at10−7 m induces EIC via NK2 receptors in the rabbit intrapulmonary artery.
A mathematical model representing the extraneuronal O-Methylating system of the perfused rat heart
TLDR
The mathematical model is able to simulate the function of the O-methylating system of the rat heart and results obtained were compared with results obtained from rat hearts perfused with 3H-isoprenaline.
Pharmacological nature of TP receptor mediated contraction in human intrapulmonary artery.
Possible involvement of endothelial leukotrienes in acetylcholine-induced contraction in rabbit coronary artery.
Bay K 8644, a voltage-sensitive calcium channel agonist, facilitates secretion of atrial natriuretic polypeptide from isolated perfused rat hearts.
Endothelium-dependent contraction and -independent relaxation induced by adenine nucleotides and nucleoside in the canine basilar artery.
TLDR
The order of agonist potency for contraction was ATP greater than ADP much greater than AMP = adenosine, but for producing relaxation the agonists were equipotent, and the relaxation in response to ATP was attenuated by removal of the endothelium.
Endothelium‐Dependent Contraction Produced by Acetylcholine and Relaxation Produced by Histamine in Monkey Basilar Arteries
An endothelium‐dependent contraction in canine mesenteric artery caused by caffeine
TLDR
It is demonstrated that caffeine causes an EDC in canine mesenteric artery and suggested that the EDCF mediating this response is probably not arachidonic acid metabolites, endothelin or superoxide, and that caffeine‐induced EDC may be due to activation of the phospholipase D pathway.
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