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COX-2 and PPAR-γ Confer Cannabidiol-Induced Apoptosis of Human Lung Cancer Cells
TLDR
In lung cancer cell lines and primary cells from a patient with lung cancer, cannabidiol elicited decreased viability associated with apoptosis and caused upregulation of COX-2 and PPAR-γ in tumor tissue and tumor regression that was reversible by GW9662. Expand
Decreased IL-10+ regulatory B cells (Bregs) in lupus nephritis patients
TLDR
The percentages of IL-10+ Bregs were significantly decreased in SLE patients, in particular those with lupus nephritis (LN), compared to HC, reflecting an impaired regulatory function. Expand
Cancer Therapeutic Insights COX-2 and PPAR-g Confer Cannabidiol-Induced Apoptosis of Human Lung Cancer Cells
The antitumorigenicmechanismof cannabidiol is still controversial. This study investigates the role of COX2 and PPAR-g in cannabidiol’s proapoptotic and tumor-regressive action. In lung cancer cellExpand