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We have participated in three open tracks of Chinese word segmentation and named entity recognition tasks of SIGHAN Bakeoff3. We take a probabilistic feature based Maximum Entropy (ME) model as our basic frame to combine multiple sources of knowledge. Our named entity recognizer achieved the highest F measure for MSRA, and word segmenter achieved the medium(More)
BACKGROUND Recently, several studies have demonstrated that two long non-coding RNAs (lncRNAs), HULC and MALAT1, may participate in hepatocellular carcinoma (HCC) development and progression. However, genetic variations in the two lncRNAs and their associations with HCC susceptibility have not been reported. In this study, we hypothesized that single(More)
BACKGROUND MiR-106b-25 cluster, hosted in intron 13 of MCM7, may play integral roles in diverse processes including immune response and tumorigenesis. A single nucleotide polymorphism (SNP), rs999885, is located in the promoter region of MCM7. METHODS We performed a case-control study including 1300 HBV-positive hepatocellular carcinoma (HCC) cases, 1344(More)
—In heterogeneous cellular networks (HCNs), the interference received at a user is correlated over time slots since it comes from the same set of randomly located BSs. This results in the correlations of link successes, thus affecting network performance. Under the assumptions of a K-tier Poisson network, strongest-candidate based BS association, and(More)
Certain pseudogenes may regulate their protein-coding cousins by competing for miRNAs and play an active biological role in cancer. However, few studies have focused on the association of genetic variations in pseudogenes with cancer prognosis. We selected six potentially functional single nucleotide polymorphisms (SNPs) in cancer-related pseudogenes, and(More)
Latent Membrane Protein 1 (LMP1) is a primary target for controlling tumorigenesis in Epstein-Barr virus related malignancies; in this study, we aimed to develop a specific antibody against the TES1 domain of the oncogenic LMP1. We screened a full human naïve Fab phage library against TES1 peptide, which consisted of C terminal-activating regions proximal(More)