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1-Palmitoyl phosphatidylcholines (1-palmitoyl PCs), in which the 2-position was occupied respectively by C10:0, C12:0, C14:0, C14:1, n-7, C16:0, C16:1, n-7, C18:0, C18:1(t), n-9, C18:1, n-9, C18:2, n-6, C18:3, N-3, C18:3, n-6, C18:3(5t,9,12), C22:0, C22:1, n-9, C22:2, n-6, C22:3, n-3, C22:4, n-6, C22:5, n-6 or C22:6, n-3 fatty acids, were studied as(More)
Linolenic acid deficiency has not been demonstrated clearly in warm blooded animals, yet circumstantial evidence suggests that n−3 fatty acids may have functions in these animals. The fact that several species of fish definitely require dietary n−3 fatty acids indicates that n−3 fatty acids have important and specific functions in these animals and suggests(More)
Rats were fed for two generations a purified, linolenic acid-deficient diet in which the only source of lipid was purified methyl linoleate. This diet contained about 38 mg linolenic acid/kg diet. Control rats were given the same diet supplemented with methyl linolenate (2,500 mg/kg diet). Male and female rats ranged in age from weanling pups to adults.(More)
The metabolism of phosphatidylethanolamine (PE) was studied in male rats fed a low methionine diet for 7 days with or without supplemental choline. Groups of animals were injected with 2-14C-ethanolamine and killed at intervals thereafter up to 72 hr. Liver phospholipids were isolated, and PE and phosphatidylcholine (PC) were separated by argentation(More)