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  • Brendan D Manning, Andrew R. Tee, M Nicole Logsdon, John Blenis, Lewis C. Cantley
  • Medicine, Biology
  • Molecular cell
  • 2002 (First Publication: 1 July 2002)
  • The S/T-protein kinases activated by phosphoinositide 3-kinase (PI3K) regulate a myriad of cellular processes. Here, we show that an approach using a combination of biochemistry and bioinformaticsContinue Reading
  • Philippe P Roux, David Shahbazian, +5 authors John Blenis
  • Biology, Medicine
  • The Journal of biological chemistry
  • 2007 (First Publication: 11 May 2007)
  • Converging signals from the mammalian target of rapamycin (mTOR) and phosphoinositide 3-kinase (PI3K) pathways are well established to modulate translation initiation. Less is known regarding theContinue Reading
  • Andrew R. Tee, Brendan D Manning, Philippe P Roux, Lewis C. Cantley, John Blenis
  • Medicine, Biology
  • Current Biology
  • 2003 (First Publication: 1 August 2003)
  • BACKGROUND Tuberous Sclerosis Complex (TSC) is a genetic disorder that occurs through the loss of heterozygosity of either TSC1 or TSC2, which encode Hamartin or Tuberin, respectively. Tuberin andContinue Reading
  • Philippe P Roux, Bryan A. Ballif, Rana Anjum, Steven P Gygi, John Blenis
  • Biology, Medicine
  • Proceedings of the National Academy of Sciences…
  • 2004 (First Publication: 14 September 2004)
  • Tuberous sclerosis complex (TSC) is a genetic disorder caused by mutations in either of the two tumor suppressor genes TSC1 or TSC2, which encode hamartin and tuberin, respectively. Tuberin andContinue Reading