• Publications
  • Influence

Claim Your Author Page
Ensure your research is discoverable on Semantic Scholar. Claiming your author page allows you to personalize the information displayed and manage your publications. Semantic Scholar automatically creates author pages based on data aggregated from public sources and our publisher partners.
  • Chunhua Wan, Xa Ma, +7 authors Junkang Jiang
  • Medicine, Chemistry
  • Toxicology and applied pharmacology
  • 2014 (First Publication: 15 December 2014)
  • Chronic exposure to excessive manganese (Mn) has been known to lead to neuronal loss and a clinical syndrome resembling idiopathic Parkinson's disease (IPD). p53 plays an integral role in theExpand
  • Jianya Zhao, Jingling Han, Junkang Jiang, Shangshi Shi, Chunhua Wan
  • Medicine, Biology
  • Brain Research
  • 2015 (First Publication: 30 July 2015)
  • Zinc is an essential nutrient that is important for normal brain development. Zinc deficiency has been linked to aberrant neurological development and functioning. However, the molecular mechanismsExpand
  • Jianbin Yang, Cheng Wang, +11 authors Qiyun Wu
  • Medicine, Chemistry
  • International immunopharmacology
  • 2015 (First Publication: 1 September 2015)
  • Perfluorooctane sulfonate (PFOS), a ubiquitous pollutant widely found in the environment and biota, can cause numerous adverse effects on human health. In recent years, PFOS's toxic effects on theExpand
  • Xiaohong Xu, Yunhua He, +8 authors Song He
  • Medicine, Biology
  • Leukemia research
  • 2016 (First Publication: 1 August 2016)
  • Previous studies have shown that chromodomain helicase/ATPase DNA binding protein 1-like gene (CHD1L) exerts its anti-apoptotic function in many solid cancers. However, its role in human multipleExpand
  • Jingling Han, Jianya Zhao, +5 authors Chunhua Wan
  • Biology, Medicine
  • Journal of neurochemistry
  • 2015 (First Publication: 1 September 2015)
  • Zinc plays an important role in the development and maintenance of central neural system. Zinc deficiency has been known to alter normal brain function, whose molecular mechanism remains largelyExpand