Jeffrey P. Cantle

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Corrigendum: Motivational, proteostatic and transcriptional deficits precede synapse loss, gliosis and neurodegeneration in the B6.Htt This Article contains a typographical error in the Methods section, under the subheading " Library construction, RNA Sequencing and RNASeq analysis " , where: " The fastq files were aligned using the default parameters of(More)
We investigated the appearance and progression of disease-relevant signs in the B6.HttQ111/+ mouse, a genetically precise model of the mutation that causes Huntington's disease (HD). We find that B6.HttQ111/+ mice are healthy, show no overt signs of central or peripheral inflammation, and no gross motor impairment as late as 12 months of age. Behaviorally,(More)
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