James J. Cortright

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Amphetamine exposure transiently increases Ca 2ϩ /calmodulin-dependent protein kinase II (CaMKII) ␣ expression in the nucleus accumbens (NAcc) shell and this persistently increases local GluA1 S831 phosphorylation and enhances behavioral responding to the drug. Here we assessed whether transiently interfering with CaMKII signaling using a dominant-negative(More)
Preston Garraghty and Joseph Steinmetz for the their comments into the design of my dissertation work, Dr. Dale Sengelaub for schooling me in the art of histology, and especially to my mentor, Dr. George Rebec, for his insights into the interpretation of my dissertation work and patience as I worked towards my degree. Second, I would like to thank a number(More)
The striatum, which receives projections from the entire cortical mantle, is highly responsive to sensorimotor activity. Because either systemic or intra-striatal injections of ascorbate (AA) influence behavior known to involve striatal circuits, it is possible that the level of striatal AA, which is known to fluctuate with behavioral activation, directly(More)
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