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2003 World Health Organization (WHO)/International Society of Hypertension (ISH) statement on management of hypertension
- J. Whitworth
- Medicine, Political ScienceJournal of hypertension
- 30 October 2003
There is evidence that specific agents have benefits for patients with particular compelling indications, and that monotherapy is inadequate for the majority of patients, and for patients without a compelling indication for a particular drug class, a low dose of diuretic should be considered for initiation of therapy.
World Health Organization (WHO) and International Society of Hypertension (ISH) risk prediction charts: assessment of cardiovascular risk for prevention and control of cardiovascular disease in low…
The WHO/ISH risk predication charts presented here, enable the prediction of future risk of heart attacks and strokes in people living in low and middle income countries, for the first time.
Total cardiovascular risk approach to improve efficiency of cardiovascular prevention in resource constrain settings.
1999 World Health Organization-International Society of Hypertension Guidelines for the management of hypertension. Guidelines sub-committee of the World Health Organization.
These guidelines represent the fourth revision of theWHO-ISH Guidelines and were finalised after presentation and discussion at the 7th WHO-ISH Meeting on Hypertension, Fukuoka, Japan, 29th Sept-1st Oct, 1998.
The effect of protein restriction on the progression of renal insufficiency.
- B. Ihle, G. Becker, J. Whitworth, R. Charlwood, P. Kincaid‐smith
- MedicineThe New England journal of medicine
- 28 December 1989
It is concluded that dietary protein restriction is effective in slowing the rate of progression of chronic renal failure.
World health organisation-international society of hypertension (WHO/ISH) hypertension guidelines.
- J. Whitworth, J. Chalmers
- Medicine, Political ScienceClinical and experimental hypertension
- 1 October 2004
The 2003 WHO/ISH Statement on Management of Hypertension focuses on key areas where new information has become available since the 1999 Guidelines, including establishment of thresholds and goals, treatment strategies and cost-effectiveness.
Effects of ACTH and Cortisol Administration on Blood Pressure, Electrolyte Metabolism, Atrial Natriuretic Peptide and Renal Function in Normal Man
- J. Connell, J. Whitworth, D. Davies, A. Lever, A. Richards, R. Fraser
- Medicine, BiologyJournal of hypertension
- 1 August 1987
The similarity of response to treatment suggests that cortisol is largely responsible for the effects of ACTH, and the respective roles of the marked increases in sodium retention, changes in fluid volume and vascular reactivity in the increases in blood pressure remain to be defined.
Cardiovascular Consequences of Cortisol Excess
- J. Whitworth, P. Williamson, G. Mangos, J. Kelly
- Medicine, BiologyVascular health and risk management
- 1 December 2005
This review analyses the relationship of cortisol excess, both locally and at tissue level, to these cardiovascular risk factors, and to putative mechanisms for hypertension, in the general population and in Cushing's syndrome.
Cushing, cortisol, and cardiovascular disease.
Cushing's syndrome of glucocorticoid excess is named after the eminent Boston neurosurgeon Harvey W. Cushing, and candidate mechanisms currently being examined include inhibition of the vasodilator nitric oxide system and increases in vasoconstrictor erythropoietin concentration.
Pressor responsiveness in corticosteroid-induced hypertension in humans.
In previous studies short-term cortisol increased cold pressor responses and the rise in forearm vascular resistance accompanying intra-arterial norepinephrine without an increase in overall resting…