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Nitric oxide induces neurotransmitter release from hippocampal slices.
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Increased striatal dopamine production from L-DOPA following selective inhibition of monoamine oxidase B by R(+)-N-propargyl-1-aminoindan (rasagiline) in the monkey.
Striatal extracellular fluid concentrations of dopamine and metabolites in response to direct striatal administration of two L-DOPA boluses administered sequentially were determined in three rhesus…
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Influence of Selective Inhibition of Monoamine Oxidase A or B on Striatal Metabolism of l‐DOPA in Hemiparkinsonian Rats
TLDR
A major role for MAO‐A in DA metabolism both intra‐ and extraneuronally in the rat striatum is indicated and is consistent with previous work on monoamine oxidase subtypes A and B.
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Regulation of striatal cyclic-3',5'-adenosine monophosphate accumulation and GABA release by glutamate metabotropic and dopamine D1 receptors.
  • J. Wang, K. Johnson
  • Biology, Chemistry
    The Journal of pharmacology and experimental…
  • 1 November 1995
TLDR
1S,3R-ACPD was determined to activate distinct mGluRs in the striatum that mediate either inhibition or activation of cAMP accumulation, with the latter effect being dependent on the activation of adenosine A2 receptors.
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Inhibition of the Phospholipase C‐Linked Metabotropic Glutamate Receptor by 2‐Amino‐3‐Phosphonopropionate Is Dependent on Extracellular Calcium
TLDR
It is suggested that incubation of hippocampal slices with AP‐3 induces a time‐dependent desensitization of the metabotropic response by a mechanism that is dependent on extracellular Ca2+.
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Arcaine and magnesium inhibition of the NMDA receptor ionophore complex: evidence for distinct voltage-dependent sites.
  • J. Wang, K. Johnson
  • Biology
    Canadian journal of physiology and pharmacology
  • 1 February 1992
TLDR
The data suggest that the polyamine antagonist binding site is distinct from either the phencyclidine/MK-801 site or the voltage-dependent channel site for magnesium, and that the site must be located in a region of the NMDA receptor ionophore complex capable of sensing transmembrane potential.
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Glutamate receptor activation induces carrier mediated release of endogenous GABA from rat striatal slices
TLDR
It is reported that in striatal slices about 17% of K+-induced endogenous GABA release was Ca2-independent and this could be blocked by a GABA transport inhibitor, indicating that excitatory amino acid agonists-induced GABA release is distinct from that induced by high K+ depolarization.
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Regulation of the NMDA receptor ionophore complex by polyamines and divalent cations
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