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Extract from Terminalia chebula Seeds Protect Against Experimental Ischemic Neuronal Damage Via Maintaining SODs and BDNF Levels
The results indicate that the repeated supplement of TCE protected neurons from ischemic damage induced by transient cerebral ischemia by maintaining SODs and BDNF levels as well as decreasing glial activation.
Effects of ADHD therapeutic agents, methylphenidate and atomoxetine, on hippocampal neurogenesis in the adolescent mouse dentate gyrus
Differences in neuronal damage and gliosis in the hippocampus between young and adult gerbils induced by long duration of transient cerebral ischemia
Sac-1004, a vascular leakage blocker, reduces cerebral ischemia—reperfusion injury by suppressing blood–brain barrier disruption and inflammation
It was shown that Sac-1004 effectively ameliorated neurological deficits and ischemic damage in a rat model of transient focal cerebral ischemia, suggesting that it is a great therapeutic candidate for stroke.
Long-Term Exercise Improves Memory Deficits via Restoration of Myelin and Microvessel Damage, and Enhancement of Neurogenesis in the Aged Gerbil Hippocampus After Ischemic Stroke
The results suggest that long-term treadmill exercise after ischemia-reperfusion can restore memory function through replacement of multiple damaged structures in the ischemic aged hippocampus.
Effects of melatonin on cognitive impairment and hippocampal neuronal damage in a rat model of chronic cerebral hypoperfusion
- Choong Hyun Lee, J. Park, J. Ahn, M. Won
- Biology, PsychologyExperimental and therapeutic medicine
- 1 April 2016
The results of the present study suggest that melatonin may attenuate CCH-induced cognitive impairment and hippocampal neuronal cell damage by decreasing oxidative stress, microglial activation and the production of pro-inflammatory cytokines in the ischemic hippocampus.
Neuroprotective effects of tanshinone I from Danshen extract in a mouse model of hypoxia-ischemia
It is suggested that tanshinone I (TsI) derived from Danshen has strong potential for neuroprotection against hypoxia-ischemic damage and may be used in research into new anti-stroke medications.
Neuroprotection of a novel synthetic caffeic acid-syringic acid hybrid compound against experimentally induced transient cerebral ischemic damage.
20 mg/kg caffeic acid-syringic acid showed a strong neuroprotective effect with an inhibition of glia activation in the hippocampal CA1 region induced by transient cerebral ischemia.
Ischemic preconditioning-induced neuroprotection against transient cerebral ischemic damage via attenuating ubiquitin aggregation