Comparison of Lipid Peroxidation and Myocardial Damage Induced by Adriamycin and 4′-Epiadriamycin in Mice
The data showing that 4′-ADM lacks a statistically significant effect in increasing heart peroxidation as compared to ADM may explain its lower myocardial toxicity.
Chagas cardiomyopathy: Europe is not spared!
The compelling arguments by which chagasic cardiomyopathy should be moved upfront in the mindset of European cardiologists, actively looked for, and appropriately recognized are reviewed.
Adriamycin-induced myocardial toxicity: new solutions for an old problem?
Relationship between oxidative stress, lipid peroxidation, and ultrastructural damage in patients with coronary artery disease undergoing cardioplegic arrest/reperfusion.
Autonomic Neuropathy and Painless Myocardial Infarction in Diabetic Patients: Histologic Evidence of Their Relationship
The authors assume that the absence of pain in diabetics with myocardial infarction could be due to a lesion of the afferent nerves that conduct pain.
Carotid Artery Stenting Protected With an Emboli Containment System
Protected carotid stenting was performed successfully and safely in this study early in the experience with cerebrovascular protection devices, and cerebral protection will likely be necessary to minimize stroke.
Prognostic impact of Chagas disease in the United States.
Amelioration of adriamycin-induced cardiotoxicity in rabbits by prenylamine and vitamins A and E.
Protection of Distal Embolization in High-Risk Patients with Acute ST-Segment Elevation Myocardial Infarction (PREMIAR).
Endomyocardial biopsies in chronic chagasic cardiomyopathy. Immunohistochemical and ultrastructural findings.
- J. Milei, R. Storino, G. F. Fernández Alonso, R. Beigelman, S. Vanzulli, V. Ferrans
- Medicine, BiologyCardiology
Clinical, immunohistochemical and ultrastructural findings in 30 endomyocardial biopsies from patients with chronic chagasic cardiomyopathy were correlated to show that the majority of the lymphocytes were T lymphocytes and associated with necrotic or degenerating myocytes.