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Early enzyme release from myocardial cells is not due to irreversible cell damage.
Energy metabolism and enzyme release of cultured adult rat heart muscle cells during anoxia.
Development of ischemia-induced damage in defined mitochondrial subpopulations.
Fatty acid-membrane interactions in isolated cardiac mitochondria and erythrocytes.
Kinetic analysis of myocardial fatty acid oxidation suggesting an albumin receptor mediated uptake process.
An index for estimation of oxygen consumption in rat heart by hemodynamic parameters.
- J. Hütter, H. Piper, P. Spieckermann
- Biology, EngineeringThe American journal of physiology
- 1 October 1985
To find a suitable index for the estimation of O2 consumption of rat hearts by use of hemodynamic parameters, isolated hearts were perfused under different working conditions and indexes containing approximations of wall stress were not found to improve the predictive ability.
Palmitate uptake in calcium tolerant, adult rat myocardial single cells--evidence for an albumin mediated transport across sarcolemma.
Myocardial fatty acid metabolism
Absence of reoxygenation damage in isolated heart cells after anoxic injury
Results indicate that leakage of cytosolic enzymes during the early stage of anoxia is due to a gradual protein release from the individual cells and is related to reversible membrane alterations, which does not induce changes considered typical of the ‘oxygen paradox’.
Acyl-carnitine effects on isolated cardiac mitochondria and erythrocytes
- M. Piper, O. Sezer, P. Schwartz, J. Hütter, C. Schweickhardt, P. Spieckermann
- BiologyBasic Research in Cardiology
- 1 March 1984
It seems questionable whether the elevated AC levels in ischemic hearts are indeed as importan for the development of membrane damage as is often supposed.