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Electroencephalograms of epileptics following sleep deprivation.
TLDR
The need exists for additional techniques that increase the incidence of abnormal discharges in epileptics without giving misleadingn activation in normal subjects and are both simple and effective. Expand
Cyanide intoxication in Macaca mulatta Physiological and neuropathological aspects
TLDR
There is no evidence for hypoxic neuronal damage of purely histotoxic type in lightly anaesthetised and spontaneously breathing M. mulatta. Expand
Cyanide intoxication in the rat: physiological and neuropathological aspects.
TLDR
It was concluded that cyanide can damage neurones only through the medium of secondary effects on circulation and respiration. Expand
Neurologic, intellectual, and psychologic sequelae following western encephalitis
TLDR
Observations on the sequelae of western encephalitis are extended to another population and to a different ecologic region to add to the knowledge of postencephalitic sequelae an estimate of the economic costs of brain damage among infants and children. Expand
Dextroamphetamine-sulfate-induced dyskinesias.
Dyskinesia developed in four patients soon after the administration of therapeutic doses of dextroamphetamine sulfate. The movements were tic-like, dystonic, and similar to those occasionallyExpand
The pathogenesis of ischaemic neuronal damage along the cerebral arterial boundary zones in Papio anubis.
TLDR
It was concluded that in a variety of clinical settings a simple EEG-based monitoring system would be optimal for the detection of an impending failure of cerebral oxygen supply. Expand
Complications of carotid manipulation
TLDR
It is reported that, occasionally, patients have spontaneous episodes of syncope because of hypersensitivity of the carotid sinus and, not until 1941 was it recorded that irreversible neurologic damage could result from this maneuver. Expand
Profound hypoxia in Papio anubis and Macaca mulatta — Physiological and neuropathological effects I. Abrupt exposure following normoxia II. Abrupt exposure following moderate hypoxia
TLDR
In the two MM with brain damage there was evidence of reduction in cerebral perfusion near the end of profound hypoxia, which contrasts with the frequent and often severe brain damage in MM after equivalent sub-atmospheric decompressions preceded by exposure to moderate altitude. Expand
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