Jéssica R. S. Alves

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BACKGROUND The human malaria parasite Plasmodium vivax infects red blood cells through a key pathway that requires interaction between Duffy binding protein II (DBPII) and its receptor on reticulocytes, the Duffy antigen/receptor for chemokines (DARC). A high proportion of P. vivax-exposed individuals fail to develop antibodies that inhibit DBPII-DARC(More)
Plasmodium vivax infects human reticulocytes through a major pathway that requires interaction between an apical parasite protein, the Duffy binding protein (region II, DBPII) and its cognate receptor on reticulocytes, the Duffy antigen receptor for chemokines (DARC). Although most people naturally exposed to P. vivax fail to develop antibo-dies that(More)
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