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  • Zong Fang Chen, Ivan S. Yuhanna, Zoya Galcheva-Gargova, Richard H Karas, Michael E. Mendelsohn, Philip W. Shaul
  • Medicine, Biology
  • The Journal of clinical investigation
  • 1999 (First Publication: 1 February 1999)
  • Estrogen is an important vasoprotective molecule that causes the rapid dilation of blood vessels by activating endothelial nitric oxide synthase (eNOS) through an unknown mechanism. In studies ofExpand
  • Chieko Mineo, Ivan S. Yuhanna, Michael J. Quon, Philip W. Shaul
  • Medicine, Biology
  • The Journal of Biological Chemistry
  • 2003 (First Publication: 14 March 2003)
  • High density lipoprotein (HDL) activates endothelial nitric-oxide synthase (eNOS), leading to increased production of the antiatherogenic molecule NO. A variety of stimuli regulate eNOS activityExpand
  • Alyson Janna Blair, Philip W. Shaul, Ivan S. Yuhanna, Patricia A. Conrad, Eric J. Smart
  • Biology, Medicine
  • The Journal of Biological Chemistry
  • 1999 (First Publication: 5 November 1999)
  • Hypercholesterolemia-induced vascular disease and atherosclerosis are characterized by a decrease in the bioavailability of endothelium-derived nitric oxide. Endothelial nitric-oxide synthase (eNOS)Expand
  • Annette M. Uittenbogaard, Philip W. Shaul, Ivan S. Yuhanna, Alyson Janna Blair, Eric J. Smart
  • Biology, Medicine
  • The Journal of Biological Chemistry
  • 2000 (First Publication: 14 April 2000)
  • Oxidized LDL (oxLDL) depletes caveolae of cholesterol, resulting in the displacement of endothelial nitric-oxide synthase (eNOS) from caveolae and impaired eNOS activation. In the present study, weExpand