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Normal range and longitudinal blood chemistry and hematology valves in juvenile and adult rheusus monkeys (Macaca mulatta).
TLDR
Two populations of rhesus monkeys, Macaca mulatta, were subjected to a series of blood chemical and hematologic evaluations, demonstrating that several conventional blood tests, viz., potassium, chloride, calcium, total bilirubin, total protein, SGOT (AST), hematocrit, and leukocyte count, varied significantly. Expand
Chronic and subchronic effects of various forms of carrageenan in rats.
TLDR
No evidence was obtained for any direct effect of the carrageenans on the liver or the gastrointestinal tract, and Parameters utilized for detecting toxicity included mortality, body weights, fecal occult blood, hematologic indices, clinical chemistry, gross and microscopic histology, and evidence of carragean storage. Expand
Monoamine oxidase inhibition.
Paternal effects of ethanol in the long-evans rat.
TLDR
Fetuses fathered by alcoholic male rats were malformed: 55% had soft-tissue anomalies and average pups weights were also reduced by paternal ethanol consumption, and no recovery in reproductive function was evident over the 21-d post-ethanol mating period. Expand
Chemically induced (streptozotocin-alloxan) diabetes mellitus in the dog. Biochemical and ultrastructural studies.
TLDR
Chemically induced diabetes mellitus in dogs is a reproducible animal model that should prove useful in studies requiring repeated experimental manipulations or sampling of biologic fluids in order to evaluate the long-term effects of different routes of delivery or preparations of insulin to control the persistent hyperglycemia. Expand
Chronic ingestion by dogs of methyl bromide-fumigated food.
STUDIES IN CARDIAC NECROSIS. II. CARDIOVASCULAR EFFECTS OF SYMPATHOMIMETIC AMINES PRODUCING CARDIAC LESIONS.
TLDR
The probable mechanisms of impairment differed among the drugs, thus confirming the pluricausal origin of drug-induced myocardial necrosis, and three mechanisms for impairment of function are proposed. Expand
STUDIES IN CARDIAC NECROSIS. I. PRODUCTION OF CARDIAC LESIONS WITH SYMPATHOMIMETIC AMINES.
TLDR
The results of these experiments indicate that many sympathomimetic amines can cause cardiac lesions and the response to isoproterenol is atypical in this respect because only isoprotserenol caused cardiac parenchymal cell necrosis. Expand
STUDIES IN CARDIAC NECROSIS. 3. METABOLIC EFFECTS OF SYMPATHOMIMETIC AMINES PRODUCING CARDIAC LESIONS.
TLDR
It is proposed that amines which cause a marked elevation in plasma NEFA also cause an accumulation of lipids in the myocardium, which can lead to the development of cardiac lesions through an ultimate mechanism which is still unknown. Expand
Autonomic responses of the circular muscles of the isolated human fallopian tube.
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