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Maternal micronutrient restriction programs the body adiposity, adipocyte function and lipid metabolism in offspring: A review
TLDR
The role of maternal micronutrients in the foetal origins of adiposity is focused mainly on, with a view to insights into the probable underlying / associated mechanisms in the metabolic pathways of endocrine related complications.
Impact of maternal chromium restriction on glucose tolerance, plasma insulin and oxidative stress in WNIN rat offspring.
TLDR
Maternal dietary CrR induced IR, impaired glucose tolerance in WNIN rat offspring and was associated with increased oxidative stress, which may predispose them to type 2 diabetes in their later life.
Chronic Maternal Dietary Chromium Restriction Modulates Visceral Adiposity
TLDR
Chronic maternal Cr restriction increased body adiposity probably due to increased stress and altered lipid metabolism in WNIN rat offspring, which may predispose them to obesity and associated diseases in later life.
Long‐term Effects of Maternal Magnesium Restriction on Adiposity and Insulin Resistance in Rat Pups
TLDR
The long‐term effects of maternal/postnatal magnesium restriction on adiposity, glucose tolerance, and insulin secretion in the offspring and the probable biochemical mechanisms associated with them are investigated.
Prenatal and perinatal zinc restriction: effects on body composition, glucose tolerance and insulin response in rat offspring
TLDR
Chronic maternal zinc restriction altered the body composition and impaired the glucose‐induced insulin secretion in the offspring and changed the plasma lipid profile among offspring of different groups.
Modulation of macronutrient metabolism in the offspring by maternal micronutrient deficiency in experimental animals.
TLDR
This review focuses on different animal models of micronutrient restriction, mimicking human situations during pregnancy and lactation that cause aberrations in macronsutrient metabolism in the offspring.
Maternal dietary chromium restriction programs muscle development and function in the rat offspring
TLDR
The results show that maternal Cr restriction in rats may irreversibly impair muscle development and glucose uptake by muscle and modulation of muscle atrophy appears to be an adaptive mechanism to preserve muscle mass in CrR offspring.
Maternal Manganese Restriction Increases Susceptibility to High-Fat Diet-Induced Dyslipidemia and Altered Adipose Function in WNIN Male Rat Offspring
TLDR
Maternal manganese restriction increased the susceptibility of the offspring to high-fat-induced adiposity, dyslipidaemia, and a proinflammatory state but did not affect their glycemic or insulin status.
Response to Comment on: Padmavathi et al. (2010) Chronic Maternal Dietary Chromium Restriction Modulates Visceral Adiposity: Probable Underlying Mechanisms. Diabetes;59:98–104
TLDR
The finding that the control diet had 1.56 mg Cr/kg diet suggests that this could be due to contributions from other dietary ingredients, and this is corroborated by the observation that Cr-restricted diet, in which Cr salt was deleted, had 0.51 mg Cr /kg diet.
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