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The Correlation between Prorocentrum donghaiense Blooms and the Taiwan Warm Current in the East China Sea - Evidence for the “Pelagic Seed Bank” Hypothesis
TLDR
Results support the hypothesis that P. donghaiense blooms develop from the population at the TWC front in the ECS, suggesting the role of the ocean current front as a seed bank to dinoflagellate blooms. Expand
14,15-Epoxyeicosatrienoic Acid Suppresses Cigarette Smoke Extract-Induced Apoptosis in Lung Epithelial Cells by Inhibiting Endoplasmic Reticulum Stress
TLDR
The data suggest that CYP2J2-derived EETs protect against CSE-induced lung injury possibly through attenuating ER stress. Expand
Promoting Effects of the Adipokine, Apelin, on Diabetic Nephropathy
TLDR
Upregulated apelin in type 2 diabetes, which may be attributed to increased fat mass, promotes angiogenesis in glomeruli to form abnormal vessels and that enhanced apelin increases permeability via upregulating the expression of VEGFR2 and Tie2 inglomerular endothelial cells. Expand
A Soluble Receptor for Advanced Glycation End-Products Inhibits Hypoxia/Reoxygenation-Induced Apoptosis in Rat Cardiomyocytes via the Mitochondrial Pathway
TLDR
It is concluded that the exogenous administration of sRAGE during H/R is involved in cardioprotection by inhibiting apoptosis via the mitochondrial pathway, which, if further confirmed in vivo, may have important clinical implications during H-R. Expand
Elevated expression of urotensin II and its receptor in diethylnitrosamine-mediated precancerous lesions in rat liver
TLDR
In DEN-induced precancerous liver lesions, the UII/UT system was up-regulated, which may contribute to the pathogenesis of liver cancer through a PKC- or ERK1/2-dependent pro-mitogenic pathway in an autocrine/paracrine manner. Expand
Urotensin-II-Mediated Reactive Oxygen Species Generation via NADPH Oxidase Pathway Contributes to Hepatic Oval Cell Proliferation
TLDR
It is indicated that UII-increased ROS production via the NADPH oxidase pathway is partially associated with activation of the PI3K/Akt and ERK cascades, accelerates G1/S transition, and contributes to cell proliferation. Expand
Time course label-free quantitative analysis of cardiac muscles of rats after myocardial infarction.
TLDR
The results provide the first experimental evidence of the proteins that are differentially expressed following myocardial infarction, using time-course label-free quantitative proteomics in vivo without ischemia-reperfusion injury or myocardIAL ischemIA. Expand
Soluble receptor for advance glycation end‐products inhibits ischemia/reperfusion‐induced myocardial autophagy via the STAT3 pathway
TLDR
The results suggest that sRAGE inhibits I/R injuries in the heart via a decrease in autophagy, a process that is dependent on STAT3 activation. Expand
Soluble receptor for advanced glycation end-products promotes angiogenesis through activation of STAT3 in myocardial ischemia/reperfusion injury
TLDR
It is demonstrated that activation of STAT3 contributed to the protective effects of sRAGE on myocardial I/R injury via promoting angiogenesis and signal transducer and activator of transcription 3 (STAT3) pathway. Expand
Soluble receptor for advanced glycation end-products enhanced the production of IFN-γ through the NF-κB pathway in macrophages recruited by ischemia/reperfusion.
TLDR
The findings of the present study indicated that sRAGE protected the heart from I/R injuries, which might be mediated by promoting infiltration and the differentiation of macrophages into M1, which would then synthesize and secrete IFN‑γ through activating the NF‑κB signaling pathway. Expand
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