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Functional and clinical evidence for NDRG2 as a candidate suppressor of liver cancer metastasis.
TLDR
Results suggest that NDRG2 can inhibit extracellular matrix-based, Rho-driven tumor cell invasion and migration and thereby play important roles in suppressing tumor metastasis in HCC.
N-terminal Arginylation Targets Endoplasmic Reticulum Chaperone BiP to Autophagy Through p62 Binding
We show that ATE1-encoded Arg-transfer RNA transferase (R-transferase) of the N-end rule pathway mediates N-terminal arginylation of multiple endoplasmic reticulum (ER)-residing chaperones, leading
Evaluation of annexin II as a potential serum marker for hepatocellular carcinoma using a developed sandwich ELISA method.
TLDR
The results suggest that expression of ANXA2 may be increased in HCC patients and may play an important role in liver cancer progression.
ESM-1 silencing decreased cell survival, migration, and invasion and modulated cell cycle progression in hepatocellular carcinoma
TLDR
This study demonstrates that ESM-1 as a potential tumor marker is overexpressed in most tissues and serum in the presence of HCC and is involved with cell survival, cell cycle progression, migration, and invasion of hepatocellular carcinoma cells.
Collagen Triple Helix Repeat Containing 1 (CTHRC1) acts via ERK-dependent induction of MMP9 to promote invasion of colorectal cancer cells
TLDR
Results demonstrate that CTHRC1 expression is elevated in human colon cancer cell lines and clinical specimens, and promotes cancer cell invasivity through ERK-dependent induction of MMP9 expression, and suggest that high levels of CTH RC1 expression are associated with poor clinical outcomes.
Up‐regulation and clinical significance of serine protease kallikrein 6 in colon cancer
Kallikrein‐related peptidase 6 (KLK6) encodes a trypsin‐like serine protease that is up‐regulated in several cancers, although the putative functions of KLK6 in cancer have not been elucidated. In
Expression of NDRG2 is related to tumor progression and survival of gastric cancer patients through Fas-mediated cell death
TLDR
Data suggest that inactivation of NDRG2 may elicit resistance against anticancer drug and Fas-mediated cell death, and case studies of gastric cancer patients indicate that N DRG2 expression may be involved in tumor progression and overall survival of the patients.
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