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  • Kevin K. Kim, Matthias Christian Kugler, +5 authors Harold A. Chapman
  • Biology, Medicine
  • Proceedings of the National Academy of Sciences
  • 2006 (First Publication: 29 August 2006)
  • Mechanisms leading to fibroblast accumulation during pulmonary fibrogenesis remain unclear. Although there is in vitro evidence of lung alveolar epithelial-to-mesenchymal transition (EMT), whetherExpand
  • Brigid L. M. Hogan, Christina E. Barkauskas, +15 authors Edward E Morrisey
  • Medicine, Biology
  • Cell stem cell
  • 2014 (First Publication: 7 August 2014)
  • Respiratory disease is the third leading cause of death in the industrialized world. Consequently, the trachea, lungs, and cardiopulmonary vasculature have been the focus of extensive investigations.Expand
  • Yongjie Wei, David A. Waltz, Navin Rao, Robert J. Drummond, Saul Rosenberg, Harold A. Chapman
  • Biology, Medicine
  • The Journal of biological chemistry
  • 1994 (First Publication: 23 December 1994)
  • Urokinase receptors, expressed on surfaces of many cell types, focus to the pericellular space plasminogen-dependent proteolysis important in matrix remodeling and cell movement. We now report thatExpand
  • Kevin K. Kim, Ying Wei, +8 authors Harold A. Chapman
  • Medicine
  • The Journal of clinical investigation
  • 2008 (First Publication: 22 December 2008)
  • Pulmonary fibrosis, in particular idiopathic pulmonary fibrosis (IPF), results from aberrant wound healing and scarification. One population of fibroblasts involved in the fibrotic process is thoughtExpand
  • Galina K. Sukhova, Guo Ping Shi, Daniel I. Simon, Harold A. Chapman, Peter Libby
  • Medicine, Biology
  • The Journal of clinical investigation
  • 1998 (First Publication: 1 August 1998)
  • Formation of the atherosclerotic intima must involve altered metabolism of the elastin-rich arterial extracellular matrix. Proteases potentially involved in these processes remain unclear. This studyExpand