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Roles of Three Histidine Kinase Genes in Hyphal Development and Virulence of the Pathogenic Fungus Candida albicans
TLDR
Results imply that in C. albicans, CaSLN1 and CaNIK1 function upstream of CaHK1 but are in distinct signal transmission pathways.
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Isolation of CaSLN1 and CaNIK1, the genes for osmosensing histidine kinase homologues, from the pathogenic fungus Candida albicans.
TLDR
SLN1 of Candida albicans was functionally cloned using an S. cerevisiae strain in which SLN1 expression was conditionally suppressed andletion analysis of the cloned gene demonstrated that the receiver domain of C.Albicans Sln1p was not necessary to rescue SLN 1-deficient S. Cerevisiae strains.
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Oligonucleotide microarray for prediction of early intrahepatic recurrence of hepatocellular carcinoma after curative resection
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Characterization of Chitin Synthase 2 of Saccharomyces cerevisiae.
TLDR
Results suggest that Asp441, Asp562, Gln601, Arg604, and Trp605 are located in the active pocket and that they function as the catalytic residues of the enzyme.
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Saccharomyces cerevisiae GNA1, an Essential Gene Encoding a Novel Acetyltransferase Involved in UDP-N-acetylglucosamine Synthesis*
TLDR
Results indicate that YFL017C specifies glucosamine-6-phosphate acetyltransferase; therefore, the gene was designated GNA1(glucosamines- 6-ph phosphateacetyl transferase), whereas bacterial phosphoglucosamine acetyl transferring activities are intrinsic in a single polypeptide.
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Comparison of gene expression profiles between hepatitis B virus- and hepatitis C virus-infected hepatocellular carcinoma by oligonucleotide microarray data on the basis of a supervised learning…
TLDR
It is suggested that HBV and HCV cause hepatocarcinogenesis by different mechanisms and provide novel tools for diagnosis and treatment of HBV- andHCV-associated HCCs.
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The Eukaryotic UDP-N-Acetylglucosamine Pyrophosphorylases
TLDR
Results demonstrate that the UAP1 genes indeed specify eukaryotic UDP-GlcNAc pyrophosphorylase and that phosphomutase reaction precedes uridyltransfer.
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Cloning of the Candida albicans homolog of Saccharomyces cerevisiae GSC1/FKS1 and its involvement in beta-1,3-glucan synthesis
TLDR
Results demonstrate that C. albicans GSC1 is the gene for a subunit of beta-1,3-glucan synthase.
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Imbalance in expression levels of insulin-like growth factor 2 and H19 transcripts linked to progression of hepatocellular carcinoma.
TLDR
It is found that imbalances in levels of IGF2 and H19 transcripts were correlated with advanced tumor stage and poor outcome in HCC patients.
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Anti-glypican 3 antibodies cause ADCC against human hepatocellular carcinoma cells.
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