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Depression uncouples brain hate circuit
This work has provided the first evidence for the involvement of the ‘hate circuit’ in depression and suggests a potential reappraisal of the key neural circuitry involved.
Complementary diffusion tensor imaging study of the corpus callosum in patients with first-episode and chronic schizophrenia.
Significant decreased fractional anisotropy values were identified in the genu of corpus callosum in patients with chronic schizophrenia, but not first-episode schizophrenia, compared with healthy controls.
Psychotic-like experiences, trauma and related risk factors among “left-behind” children in China
Psychotic-like experiences and correlation with childhood trauma and other socio-demographic factors: A cross-sectional survey in adolescence and early adulthood in China
Abnormally increased and incoherent resting-state activity is shared between patients with schizophrenia and their unaffected siblings
White matter integrity deficit in treatment-naïve adult patients with major depressive disorder.
- X. Ouyang, H. Tao, Z. Xue
- Psychology, MedicineEast Asian archives of psychiatry : official…
- 1 March 2011
Findings support the notion that deficit of white matter integrity in the fronto-limbic system may be the neural substrate of major depressive disorder.
Opposite Effective Connectivity in the Posterior Cingulate and Medial Prefrontal Cortex between First-Episode Schizophrenic Patients with Suicide Risk and Healthy Controls
The hyper-connectivity from the left posterior cingulate cortex to the left medial prefrontal cortex may suggest that the abnormal effective connectivity was associated with risk for schizophrenia.
Changes of psychotic-like experiences and their association with anxiety/depression among young adolescents before COVID-19 and after the lockdown in China
Inefficient DMN Suppression in Schizophrenia Patients with Impaired Cognitive Function but not Patients with Preserved Cognitive Function
These findings provide the first direct evidence that disrupted DMN activity only exists in schizophrenia patients with impaired cognitive function, supporting the specific neuro-pathological role of inefficient DMN suppression in cognitive deficits of first-episode schizophrenia.