• Publications
  • Influence
The morphology of globus pallidus projection neurons in the rat: an intracellular staining study
The morphology of 23 intracellularly stained projection neurons of rat globus pallidus (GP) was studied in light microscopic preparations. The somatic size of these projection neurons was highlyExpand
  • 266
  • 32
Mn and Mg influxes through Ca channels of motor nerve terminals are prevented by verapamil in frogs
At frog neuromuscular junctions immersed in solutions containing 0.5 mM Mn2+, verapamil (40 microM) reduced the increase in miniature end-plate potential (MEPP) frequency produced by tetanicExpand
  • 238
  • 14
Anatomy and Physiology of the Subthalamic Nucleus: A Driving Force of the Basal Ganglia
The subthalamic (STh) nucleus is located between the zona incerta dorsally and the cerebral peduncle ventrally. The morphological features of STh neurons have been studied extensively at the lightExpand
  • 203
  • 5
Intracellular study of rat entopeduncular nucleus neurons in an in vitro slice preparation: electrical membrane properties
Electrical properties of rat entopeduncular nucleus (EP) neurons were studied in vitro using slice preparations. Of 108 EP neurons recorded, 104 were classified into two types based on their membraneExpand
  • 61
  • 5
Time course and magnitude of effects of changes in tonicity on acetylcholine release at frog neuromuscular junction.
1. The time course for the changes in miniature end-plate potential (min epp) frequency and in epp amplitude produced by alterations in the tonicity of the Ringer at the frog neuromuscular junctionExpand
  • 58
  • 3
Tetanic stimulation increases the frequency of miniature end-plate potentials at the frog neuromuscular junction in Mn2+-, CO2+-, and Ni2+-saline solutions
The effects of Mn2+, Co2+, and of Ni2+ on quantal acetylcholine (ACh) release have been studied with conventional microelectrode techniques. Increasing the [Co2+]0 or [Ni2+]0 (in the absence ofExpand
  • 56
  • 1
Effects of the ionophore X‐537A on acetylcholine release at the frog neuromuscular junction.
1. X‐537A is an ionophore that can carry cations across cell membranes. We studied its effects on spontaneous and stimulated quantal acetylcholine (ACh) release at the frog neuromuscular junction. 2.Expand
  • 62
Calcium ionophore X-537A increases spontaneous and phasic quantal release of acetylcholine at frog neuromuscular junction
TRANSMITTER release from nerve endings is believed to be triggered by the influx of Ca2+, which is thought to enter the terminal as a result of an increase in conductance produced by the actionExpand
  • 52