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Broadly Altered Gene Expression in Blood Leukocytes in Essential Hypertension Is Absent During Treatment
Abstract—We assessed whether large-scale expression profiling of leukocytes of patients with essential hypertension reflects characteristics of systemic disease and whether such changes areExpand
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Regulation of gene expression by dietary Ca2+ in kidneys of 25-hydroxyvitamin D3-1 alpha-hydroxylase knockout mice.
BACKGROUND Pseudovitamin D deficiency rickets (PDDR) is an autosomal disease, characterized by undetectable levels of 1,25-dihydroxyvitamin D3 (1,25(OH)2D3), rickets and secondaryExpand
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Role of Circulating Karyocytes in the Initiation and Progression of Atherosclerosis
Cardiovascular disease is still hard to predict in an individual. The main focus in cardiovascular research has been on endothelial cells and vascular smooth muscle cells of the vessel wall and theirExpand
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Enhanced Angiotensin II type 1 receptor expression in leukocytes of patients with chronic kidney disease.
Previously, we demonstrated increased Angiotensin II type I receptor expression in leukocytes from patients with untreated, but not in treated, essential hypertension (essential hypertension). WeExpand
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Myofibroblast progenitor cells are increased in number in patients with type 1 diabetes and express less bone morphogenetic protein 6: a novel clue to adverse tissue remodelling?
Aims/hypothesisGrowth factor imbalance and endothelial progenitor cell dysfunction are well-known elements of the inappropriate response to injury in human and experimental diabetes. We hypothesisedExpand
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An unusual case of abdominal arterial thrombosis in a young woman using desmopressin.
We report an unusual case of severe abdominal arterial thrombosis in a young woman using oral desmopressin. Only a few cases with cerebrovascular accidents and coronary syndromes have been describedExpand
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Gene expression of energy and protein metabolism in hearts of hypertensive nitric oxide- or GSH-depleted mice.
Hypertension demands cardiac synthetic and metabolic adaptations to increased afterload. We studied gene expression in two models of mild hypertension without overt left ventricular hypertrophy usingExpand
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