Giorgios E Kapotsis

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BACKGROUND Several studies have implicated a role of inflammation in the pathogenesis of lung damage in idiopathic pulmonary fibrosis (IPF). Parenchymal lung damage leads to defects in mechanics and gas exchange and clinically manifests with exertional dyspnea. Investigations of inflammatory cells in IPF have shown that eosinophils, neutrophils and CD8+ TLs(More)
The term Pulmonary-renal syndrome refers to the combination of diffuse alveolar haemorrhage and rapidly progressive glomerulonephritis. A variety of mechanisms such as those involving antiglomerular basement membrane antibodies, antineutrophil cytoplasm antibodies or immunocomplexes and thrombotic microangiopathy are implicated in the pathogenesis of this(More)
A 33-yr-old, Caucasian male, smoker (40 pack-yr) presented to the current authors9 hospital complaining of a painful and swollen right breast, which had already lasted a few weeks. He had poor oral hygiene, had been subject to several teeth extractions over the previous 2 yrs and had sporadically used oral antibiotics. He denied fever, cough and shortness(More)
A 63-yr-old male presented with a 20-day history of fever with chills, rigors, sweats and fatigue. He resided in the countryside and there was no history of recent travel to other countries. He had a past medical history of idiopathic pulmonary fibrosis (usual interstitial pneumonia confirmed by surgical biopsy) and the last 2 yrs he was taking daily(More)
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