Gastone T Matioli

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A mechanism for converting human chromatid 17 into its isoforms-isochromatid 17q and isochromosome X17q--is discussed. The frequency of these isoforms in a subset of Ph+ chronic myeloid leukemia patients is traceable to singularities of the nuclear envelope of myeloid cells. When statistically interpreted, loss of tumor suppressor gene (p53) seems less(More)