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Acetylcholine, GABA and glutamate induce ionic currents in cultured antennal lobe neurons of the honeybee, Apis mellifera
The honeybee, Apis mellifera, is a valuable model system for the study of olfactory coding and its learning and memory capabilities. In order to understand the synaptic organisation of olfactoryExpand
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  • Open Access
T-Type Calcium Channel Inhibition Underlies the Analgesic Effects of the Endogenous Lipoamino Acids
Lipoamino acids are anandamide-related endogenous molecules that induce analgesia via unresolved mechanisms. Here, we provide evidence that the T-type/Cav3 calcium channels are importantExpand
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  • Open Access
Study of nicotinic acetylcholine receptors on cultured antennal lobe neurones from adult honeybee brains
In insects, acetylcholine (ACh) is the main neurotransmitter, and nicotinic acetylcholine receptors (nAChRs) mediate fast cholinergic synaptic transmission. In the honeybee, nAChRs are expressed inExpand
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  • Open Access
Homomeric RDL and heteromeric RDL/LCCH3 GABA receptors in the honeybee antennal lobes: two candidates for inhibitory transmission in olfactory processing.
gamma-Aminobutyric acid (GABA)-gated chloride channel receptors are abundant in the CNS, where their physiological role is to mediate fast inhibitory neurotransmission. In insects, this inhibitoryExpand
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  • Open Access
Regulation of T-type calcium channels: signalling pathways and functional implications.
T-type calcium channels (T-channels) contribute to a wide variety of physiological functions, especially in the cardiovascular and nervous systems. Recent studies using knock-out mouse models haveExpand
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Akt regulates L-type Ca2+ channel activity by modulating Cavα1 protein stability
The insulin IGF-1–PI3K–Akt signaling pathway has been suggested to improve cardiac inotropism and increase Ca2+ handling through the effects of the protein kinase Akt. However, the underlyingExpand
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  • Open Access
Selective Inhibition of T-Type Calcium Channels by Endogenous Lipoamino Acids
T-type calcium channels, i.e. Cav3.1, Cav3.2 and Cav3.3 channels, have important roles in cell excitability and calcium signalling and contribute to a wide variety of physiological functionsExpand